Mini Sample Human TNF-B Beta (Tumor Necrosis Factor Beta) ELISA Kit

Size

£128.70

Was £143.00

SKU:: E-MSEL-H0042
Additional Names:: LT,LTA,TNFB,TNFSF1,TNF Superfamily,Member 1,Lymphotoxin Alpha
Application:: ELISA
Physical State:: Other Biological Fluid, Plasma, Serum
Species Reactivity:: Human
Storage Conditions:: 2-8[o]C
Supplier:: Elabscience
Format:: Colorimetric method,ELISA,Sandwich
Uniprot:: P01374
Synonyms:: LT;LT-alpha;lymphotoxin-alpha;TNF superfamily, member 1;TNF-beta;TNFB;TNFSF1;TNLG1E;truncated lymphotoxin alpha;tumor necrosis factor beta;tumor necrosis factor ligand 1E;tumor necrosis factor ligand superfamily member 1
Extra Details:: Human TNF-B Beta (tumor necrosis factor B Beta, also known as lymphotoxin A Alpha, LT-A Alpha) is a member of the TNF family with a molecular weight of about 22kDa and a high degree of homology (about 73% amino acid homology) with TNF molecules in mice and rats. TNF-B Beta is produced primarily by activated T and B cells and acts by binding to its receptors TNFR1 and TNFR2. NF-B Beta plays an important role in inflammation and immune responses. Studies have shown that TNF-B Beta can activate the NF-K KappaB signaling pathway, thereby promoting the expression of inflammatory factors . In addition, TNF-B Beta is also involved in the regulation of cell apoptosis, proliferation, differentiation and lipid metabolism. In studies of osteoarthritis, TNF-B Beta has been found to promote apoptosis and inflammatory response of chondrocytes by activating the NF-K KappaB-dependent inflammatory environment. Human TNF-B Beta (tumor necrosis factor B Beta, also known as lymphotoxin-A Alpha, LT-A Alpha) is a member of the TNF family with a molecular weight of approximately 22kDa. The TNF molecule has a high homology (about 73% amino acid homology) with mice and rats. TNF-B Beta is produced primarily by activated T and B cells and acts by binding to its receptors TNFR1 and TNFR2. NF-B Beta plays an important role in inflammation and immune responses. Studies have shown that TNF-B Beta can activate the NF-K KappaB signaling pathway, thereby promoting the expression of inflammatory factors . In addition, TNF-B Beta is also involved in the regulation of cell apoptosis, proliferation, differentiation and lipid metabolism. In studies of osteoarthritis, TNF-B Beta has been found to promote apoptosis and inflammatory responses in chondrocytes by activating the NF-K KappaB-dependent inflammatory environment

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